BMI-1 activation is crucial in hTERT-induced epithelial–mesenchymal transition of oral epithelial cells
✍ Scribed by Qiao, Bin; Chen, Zhifeng; Hu, Fengchun; Tao, Qian; Lam, Alfred K.
- Book ID
- 123429578
- Publisher
- Elsevier Science
- Year
- 2013
- Tongue
- English
- Weight
- 765 KB
- Volume
- 95
- Category
- Article
- ISSN
- 0014-4800
No coin nor oath required. For personal study only.
📜 SIMILAR VOLUMES
## Abstract Cell morphology dictates response to a wide variety of stimuli, controlling cell metabolism, differentiation, proliferation, and death. Epithelial‐mesenchymal transition (EMT) is a developmental process in which epithelial cells acquire migratory characteristics, and in the process conv
## Abstract Epithelial‐mesenchymal transition (EMT) is a process occurring during both embryogenesis and early stages of invasive cancer. Epithelial cells that undergo EMT become more migratory and invasive with a mesenchymal morphology. Herein we assess EMT induction in a mouse mammary epithelial
## Abstract Constitutive activation of the MAP kinase kinase MEK1 induces oncogenic transformation in intestinal epithelial cells. Loss of cell–cell adhesion followed by the dissociation of epithelial structures is a prerequisite for increased cell motility and tumor invasion. This phenotypic switc