## Abstract Our previous study demonstrates that Bcr‐Abl fusion oncogene frequently found in chronic myeloid leukemia (CML) cells can up‐regulate Skp2 expression via transcriptional activation. However, Bcr‐Abl also modulates Skp2 protein stability in these cells. Treatment of Bcr‐Abl kinase inhibi
✦ LIBER ✦
Bcr-Abl stabilizes β-catenin in chronic myeloid leukemia through its tyrosine phosphorylation
✍ Scribed by Coluccia, Addolorata Maria Luce; Vacca, Angelo; Duñach, Mireia; Mologni, Luca; Redaelli, Sara; Bustos, Victor H; Benati, Daniela; Pinna, Lorenzo A; Gambacorti-Passerini, Carlo
- Book ID
- 110031865
- Publisher
- Nature Publishing Group
- Year
- 2007
- Tongue
- English
- Weight
- 541 KB
- Volume
- 26
- Category
- Article
- ISSN
- 0261-4189
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