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Autoimmune epididymoorchitis is essential to the pathogenesis of male-specific spondylarthritis in HLA–B27–transgenic rats

✍ Scribed by Joel D. Taurog; Claudia Rival; Leonie M. van Duivenvoorde; Nimman Satumtira; Martha L. Dorris; Margaret Sun; John M. Shelton; James A. Richardson; F. Kent Hamra; Robert E. Hammer; Kenneth S. K. Tung

Book ID
112002761
Publisher
John Wiley and Sons
Year
2012
Tongue
English
Weight
617 KB
Volume
64
Category
Article
ISSN
0004-3591

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HLA–B27 up-regulation causes accumulation of misfolded heavy chains and correlates with the magnitude of the unfolded protein response in transgenic rats: Implications for the pathogenesis of spondylarthritis-like disease
✍ Matthew J. Turner; Monica L. DeLay; Shuzhen Bai; Erin Klenk; Robert A. Colbert 📂 Article 📅 2006 🏛 John Wiley and Sons 🌐 English ⚖ 213 KB 👁 1 views

Objective. HLA-B27 is implicated in the pathogenesis of spondylarthritis (SpA), yet the molecular mechanisms are incompletely defined. HLA-B27 misfolding has been associated with endoplasmic reticulum stress and activation of the unfolded protein response (UPR) in macrophages from HLA-B27/human ␤ 2