Auditory middle-latency responses in patients with localized and non-localized lesions of the central nervous system
β Scribed by Yumiko Kaseda; Shozo Tobimatsu; Takato Morioka; Motohiro Kato
- Publisher
- Springer
- Year
- 1991
- Tongue
- English
- Weight
- 657 KB
- Volume
- 238
- Category
- Article
- ISSN
- 0340-5354
No coin nor oath required. For personal study only.
β¦ Synopsis
Auditory middle-latency responses were recorded in 45 neurological patients. In 4 patients with localized lesions in the thalamus, or subcortical white matter, the component Na was attenuated or delayed at T3, Cz and T4, exclusively on stimulation of the ear contralateral to the lesion. In 5 patients with temporal lobe lesions with receptive aphasia, the amplitude of component Pa was decreased on the lesion side (T3), regardless of the side of stimulation. These findings suggest that Na is generated from a subcortical structure and Pa is generated from the temporal cortex. In 25 patients with diffuse CNS lesions, V-Na interpeak latency was prolonged in 2 cases.
π SIMILAR VOLUMES
By analogy with other infections of the central nervous system (CNS), it is believed that schistosomal myeloradiculopathy (SMR) is an entity that may involve a mild-to-moderate degree of impairment of the blood-brain barrier along with intrathecal synthesis of antibodies. The first of these aspects
## Abstract Inflammatory cells enter the CNS and target myelin in multiple sclerosis (MS) and experimental allergic encephalomyelitis (EAE), a model of MS, and inflammation is thought to induce stress responses in the CNS. Protein kinase R (PKR) and eukaryotic initiation factorβ2Ξ± (eIF2Ξ±) undergo p