I n the absence of oxygen a nerve gradually loses its ability to conduct; when the gas is made available conduction promptly returns. The lack of oxygen might condition failure of conduction via several mechanisms : 1) decrease in energyyielding reactions with a consequent breakdown in the functiona
Asphyxial potassium loss of mammalian nerve
β Scribed by Van Harreveld, A.
- Publisher
- Wiley (John Wiley & Sons)
- Year
- 1951
- Tongue
- English
- Weight
- 371 KB
- Volume
- 38
- Category
- Article
- ISSN
- 0095-9898
No coin nor oath required. For personal study only.
β¦ Synopsis
Fenn ('40) reported that frog nerve immersed for 3 hours in Ringer's solution does not lose more potassium to the surrounding fluid in tlie absence of 0,' than when oxygenated.
However in a more recent investigation, Fenn and Gerscliman ('50) found less potassium in frog nerve asphyxiated for
π SIMILAR VOLUMES
It is of interest in the light of recent experiments showing the relatively high permeability of muscle to cattiom to discover whether the high potassium content of muscle is maintained by some energy producing system within the fibers. Fenn ('40) reviews the literature and concludes that potassium
Isaacs' syndrome (acquired neuromyotonia) is an antibody-mediated potassium channel disorder (channelopathy). The target channel proteins of the antigens are voltage-gated potassium channels (VGKCs), especially dendrotoxin-sensitive fast potassium channels. The suppression of voltage-gated outward K
The lack of axonal regeneration in the adult mammalian CNS is due to both unfavorable environmental glial factors and the intrinsic neuronal state. Inhibitors associated with myelin and the glial scar have been extensively studies and it has been shown that neutralizing at least some of the inhibito