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Apoptotic Cell Death and Impairment of L-Type Voltage-Sensitive Calcium Channel Activity in Rat Cerebellar Granule Cells Treated with the Prion Protein Fragment 106–126

✍ Scribed by Stefano Thellung; Tullio Florio; Valentina Villa; Alessandro Corsaro; Sara Arena; Carolina Amico; Mauro Robello; Mario Salmona; Gianluigi Forloni; Orso Bugiani; Fabrizio Tagliavini; Gennaro Schettini

Book ID
115641532
Publisher
Elsevier Science
Year
2000
Tongue
English
Weight
281 KB
Volume
7
Category
Article
ISSN
0969-9961

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Prion protein fragment 106–126 induces a
Prion protein fragment 106–126 induces apoptotic cell death and impairment of L-type voltage-sensitive calcium channel activity in the GH3 cell line
✍ Tullio Florio; Stefano Thellung; Carolina Amico; Mauro Robello; Mario Salmona; O 📂 Article 📅 1998 🏛 John Wiley and Sons 🌐 English ⚖ 358 KB 👁 1 views

The prion diseases are transmissible neurodegenerative pathologies characterized by the accumulation of altered forms of the prion protein (PrP), termed PrP Sc , in the brain. Previous studies have shown that a synthetic peptide homologous to residues 106-126 of PrP (PrP 106-126) maintains many char