Susceptibility genes for human diseases (e.g., cancer and atherosclerosis) increase disease risk by altering the metabolic activation of exogenous (e.g., carcinogens) and endogenous (e.g., cholesterol) compounds. The function of these genes, and subsequent risk, can be adversely affected by polymorp
Apolipoprotein E alleles in childhood-onset schizophrenia
β Scribed by Fernandez, Thomas; Yan, Wen Liang; Hamburger, Susan; Rapoport, Judith L.; Saunders, Ann M.; Schapiro, Mark; Ginns, Edward I.; Sidransky, Ellen
- Publisher
- John Wiley and Sons
- Year
- 1999
- Tongue
- English
- Weight
- 8 KB
- Volume
- 88
- Category
- Article
- ISSN
- 0148-7299
- DOI
- 10.1002/(sici)1096-8628(19990416)88:2<211::aid-ajmg20>3.0.co;2-m
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