𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Analysis of the gap-related domain of the neurofibromatosis type 1 (NF1) gene in childhood brain tumors

✍ Scribed by Wolfram G. Scheurlen; Leonore Senf


Publisher
John Wiley and Sons
Year
1995
Tongue
French
Weight
832 KB
Volume
64
Category
Article
ISSN
0020-7136

No coin nor oath required. For personal study only.


πŸ“œ SIMILAR VOLUMES


Characterization of the somatic mutation
✍ Meena Upadhyaya; Song Han; Claudia Consoli; Elisa Majounie; Martin Horan; Nick S πŸ“‚ Article πŸ“… 2004 πŸ› John Wiley and Sons 🌐 English βš– 307 KB πŸ‘ 1 views

## Communicated by Haig H. Kazazian One of the main features of neurofibromatosis type 1 (NF1) is benign neurofibromas, 10-20% of which become transformed into malignant peripheral nerve sheath tumors (MPNSTs). The molecular basis of NF1 tumorigenesis is, however, still unclear. Ninety-one tumors

Alternative splicing of neurofibromatosi
✍ Hiroshi Mochizuki; Toru Nishi; Janet M. Bruner; Polly S. Y. Lee; Victor A. Levin πŸ“‚ Article πŸ“… 1992 πŸ› John Wiley and Sons 🌐 English βš– 496 KB

## Abstract The neurofibromatosis type 1 (__NF__1) gene encodes a 360‐residue region showing significant homology to the catalytic domains of both mammalian GTPase‐activating protein (GAP) and yeast IRA protein. The product of the GAP‐related domain of the __NF__1 gene (__NF__1‐GRD) has been shown