Analysis of p53 and BAX mutations, loss of heterozygosity, p53 and BCL2 expression and apoptosis in basal cell carcinoma in Korean patients
β Scribed by S. Cho; J-H. Hahm; Y-S. Hong
- Publisher
- John Wiley and Sons
- Year
- 2001
- Tongue
- English
- Weight
- 339 KB
- Volume
- 144
- Category
- Article
- ISSN
- 0007-0963
No coin nor oath required. For personal study only.
π SIMILAR VOLUMES
BACKGROUND. Bax, Bcl-2, and p53 proteins are involved in the regulation of apoptosis and have been reported to correlate with prognosis in several tumor types. ## METHODS. Bax, Bcl-2, p53, and the level of spontaneous apoptosis were evaluated in formalin fixed, paraffin embedded pretreatment speci
## Abstract ## BACKGROUND The histologic subtype of a basal cell carcinoma (BCC) may be an important factor for the success of a certain treatment modality. In the current article, the authors report recurrence rates among patients with BCC after superficial radiotherapy as well as Bclβ2 and p53 e
## Abstract ## Background. The p53 protein, a wellβknown tumor suppressor that functions primarily as a transcription factor, initiates cell cycle arrest and apoptosis after genotoxic stress. The antiapoptotic regulator Bclβ2 is a downstream modulator of p53βinduced apoptosis. Loss of function of