𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Analysis of genotoxicity and the carcinogenic mode of action for ortho-phenylphenol

✍ Scribed by David Brusick

Publisher
John Wiley and Sons
Year
2005
Tongue
English
Weight
204 KB
Volume
45
Category
Article
ISSN
0893-6692

No coin nor oath required. For personal study only.

πŸ“œ SIMILAR VOLUMES

An evaluation of the mode of action fram
An evaluation of the mode of action framework for mutagenic carcinogens case study: Cyclophosphamide
✍ Nancy McCarroll; Nagalakshmi Keshava; Michael Cimino; Margaret Chu; Kerry Dearfi πŸ“‚ Article πŸ“… 2008 πŸ› John Wiley and Sons 🌐 English βš– 246 KB πŸ‘ 1 views

## Abstract In response to the 2005 revised US Environmental Protection Agency (EPA) Cancer Guidelines, a Risk Assessment Forum's Technical Panel has devised a strategy in which genetic toxicology data combined with other information are assessed to determine whether a carcinogen operates through a

Identification of a genotoxic mechanism
Identification of a genotoxic mechanism for the carcinogenicity of the environmental pollutant and suspected human carcinogen o-anisidine
✍ Marie StiborovΓ‘; MarkΓ©ta MikΕ‘anovΓ‘; Miroslav Ε ulc; Helena RΓ½dlovΓ‘; Heinz H. Schm πŸ“‚ Article πŸ“… 2005 πŸ› John Wiley and Sons 🌐 French βš– 301 KB πŸ‘ 1 views

2-methoxyaniline (o-anisidine) is an industrial and environmental pollutant and a bladder carcinogen for rodents. The mechanism of its carcinogenicity was investigated with 2 independent methods, 32 P-postlabeling and 14 C-labeled o-anisidine, to show that o-anisidine binds covalently to DNA in vitr

An evaluation of the mode of action fram
An evaluation of the mode of action framework for mutagenic carcinogens case study II: Chromium (VI)
✍ Nancy McCarroll; Nagalakshmi Keshava; Jonathan Chen; Gregory Akerman; Andrew Kli πŸ“‚ Article πŸ“… 2009 πŸ› John Wiley and Sons 🌐 English βš– 434 KB πŸ‘ 1 views

## Abstract In response to the 2005 revised U.S Environmental Protection Agency's (EPA) Cancer Guidelines, a strategy is being developed to include all mutagenicity and other genotoxicity data with additional information to determine whether the initiating step in carcinogenesis is through a mutage