𝔖 Bobbio Scriptorium
✦   LIBER   ✦

An update on the role of free radicals and antioxidant defense in human disease

✍ Scribed by G. Vendemiale; I. Grattagliano; E. Altomare

Publisher
Springer
Year
1999
Tongue
English
Weight
898 KB
Volume
29
Category
Article
ISSN
0940-5437

No coin nor oath required. For personal study only.

πŸ“œ SIMILAR VOLUMES

The activity of recombinant human neurog
The activity of recombinant human neuroglobin as an antioxidant and free radical scavenger
✍ Weiguang Li; Yonghong Wu; Changhong Ren; Yiming Lu; Yan Gao; Xiaofei Zheng; Chen πŸ“‚ Article πŸ“… 2010 πŸ› John Wiley and Sons 🌐 English βš– 381 KB

## Abstract Free radicals are by‐products of metabolism and exist in a homeostasis between generation and scavenging __in vivo__. Excessive free radicals cause various diseases, including nervous system diseases. Neuroglobin (Ngb), a nervous system‐specific oxygen‐binding protein, has been suggeste

Protein glutathionylation in human centr
Protein glutathionylation in human central nervous system: Potential role in redox regulation of neuronal defense against free radicals
✍ Marco Sparaco; Laura Maria Gaeta; Giulia Tozzi; Enrico Bertini; Anna Pastore; Al πŸ“‚ Article πŸ“… 2006 πŸ› John Wiley and Sons 🌐 English βš– 787 KB

## Abstract Neuronal defense against free radicals is mediated primarily by the glutathione system. A cerebral defect of this system gives rise to the oxidative stress occurring in some neurological diseases. Glutathione provides a means of regulating protein function by glutathionylation, consisti

Effects of neonatal quinalphos exposure
Effects of neonatal quinalphos exposure and subsequent withdrawal on free radical generation and antioxidative defenses in developing rat brain
✍ Alka Gupta; Amita Gupta; Girja S. Shukla πŸ“‚ Article πŸ“… 1998 πŸ› John Wiley and Sons 🌐 English βš– 59 KB πŸ‘ 2 views

Ten-day-old rat pups were given quinalphos (QP, 0.5 mg kg ؊1 ) orally up to postnatal days (PND) 21 or 45. A group of rats exposed to QP was withdrawn from the treatment at PND 21 and was killed at PND 45 for the withdrawal study. Acetylcholinesterase decreased in the brain and blood after QP exposu

NKX2–5: an update on this hypermutable h
NKX2–5: an update on this hypermutable homeodomain protein and its role in human congenital heart disease (CHD)
✍ Stella Marie Reamon-Buettner; Juergen Borlak πŸ“‚ Article πŸ“… 2010 πŸ› John Wiley and Sons 🌐 English βš– 415 KB

Congenital heart disease (CHD) is among the most prevalent and fatal of all birth defects. Deciphering its causes, however, is complicated, as many patients affected by CHD have no family history of the disease. There is also widespread heterogeneity of cardiac malformations within affected individu