✦ LIBER ✦

An intracellular protein that binds amyloid-β peptide and mediates neurotoxicity in Alzheimer's disease

✍ Scribed by Du Yan, Shi; Fu, Jin; Soto, Claudio; Chen, Xi; Zhu, Huaijie; Al-Mohanna, Futwan; Collison, Kate; Zhu, Aiping; Stern, Eric; Saido, Takaomi; Tohyama, Masaya; Ogawa, Satoshi; Roher, Alex; Stern, David

Book ID: 109800042
Publisher: Nature Publishing Group
Year: 1997
Tongue: English
Weight: 548 KB
Volume: 389
Category: Article
ISSN: 0028-0836
DOI: 10.1038/39522

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Reverse relationship between β-amyloid p

Reverse relationship between β-amyloid precursor protein and β-amyloid peptide plaques in Down’s syndrome versus sporadic/familial Alzheimer’s disease

✍ Rupert Egensperger; Sascha Weggen; Nobua Ida; Gerd Multhaup; Ralf Schnabel; Konr 📂 Article 📅 1999 🏛 Springer-Verlag 🌐 English ⚖ 260 KB

Cerebrospinal fluid amyloid β peptide pa

Cerebrospinal fluid amyloid β peptide patterns in Alzheimer's disease patients and nondemented controls depend on sample pretreatment: Indication of carrier-mediated epitope masking of amyloid β peptides

✍ Mirko Bibl; Hermann Esselmann; Markus Otto; Piotr Lewczuk; Lukas Cepek; Eckart R 📂 Article 📅 2004 🏛 John Wiley and Sons 🌐 English ⚖ 108 KB

Fenton chemistry and oxidative stress me

Fenton chemistry and oxidative stress mediate the toxicity of the β-amyloid peptide in a Drosophila model of Alzheimer’s disease

✍ Thomas Rival; Richard M. Page; Dhianjali S. Chandraratna; Timothy J. Sendall; Ed 📂 Article 📅 2009 🏛 John Wiley and Sons 🌐 English ⚖ 617 KB

Acetyl-L-carnitine-induced up-regulation

Acetyl-L-carnitine-induced up-regulation of heat shock proteins protects cortical neurons against amyloid-beta peptide 1–42-mediated oxidative stress and neurotoxicity: Implications for Alzheimer's disease

✍ Hafiz Mohmmad Abdul; Vittorio Calabrese; Menotti Calvani; D. Allan Butterfield 📂 Article 📅 2006 🏛 John Wiley and Sons 🌐 English ⚖ 636 KB

Alzheimer's disease (AD) is a progressive neurodegenerative disorder characterized by loss of memory and cognition and by senile plaques and neurofibrillary tangles in brain. Amyloid-beta peptide, particularly the 42-amino-acid peptide (Abeta(1-42)), is a principal component of senile plaques and is

Amyloid precursor protein cytoplasmic do

Amyloid precursor protein cytoplasmic domain with phospho-Thr668 accumulates in Alzheimer’s disease and its transgenic models: a role to mediate interaction of Aβ and tau

✍ Ryong-Woon Shin; Koichi Ogino; Alfredo Shimabuku; Takao Taki; Hanae Nakashima; T 📂 Article 📅 2007 🏛 Springer-Verlag 🌐 English ⚖ 587 KB