## Abstract Alzheimer disease (AD) is characterized neuropathologically by neurofibrillary tangles and senile plaques. A key component of plaques is Aβ, a polypeptide derived from Aβ‐precursor protein (APP) through proteolytic cleavage catalyzed by β and γ‐secretase. We hypothesized that sequence v
✦ LIBER ✦
Alzheimer disease: Joining the dots between APOE ε4 and late-onset Alzheimer disease via integrative genomics
✍ Scribed by Kingwell, Katie
- Book ID
- 120986424
- Publisher
- Nature Publishing Group
- Year
- 2013
- Tongue
- English
- Weight
- 109 KB
- Volume
- 9
- Category
- Article
- ISSN
- 1759-4758
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