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Alterations in activities of cyclic nucleotide systems and in β-adrenergic receptor-mediated activation of cyclic amp-dependent protein kinase during progression and regression of isoproterenol-induced cardiac hypertrophy

✍ Scribed by Tse, James; Brackett, Nancy L.; Kuo, J.F.


Book ID
122946555
Publisher
Elsevier Science
Year
1978
Tongue
English
Weight
870 KB
Volume
542
Category
Article
ISSN
0304-4165

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## Abstract The following study was undertaken to elucidate the cytoskeletal phenotype of neonatal rat cardiac fibroblasts (NNCF) and the signaling pathways coupled to β‐adrenergic receptor stimulated DNA synthesis. The cytoskeletal proteins vimentin, and smooth muscle α‐actin were detected in NNCF