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Alpha-1-antitrypsin-deficient phenotype is not maintained by segregation distortion

✍ Scribed by B. K. Suarez; J. A. Pierce


Publisher
John Wiley and Sons
Year
1983
Tongue
English
Weight
401 KB
Volume
62
Category
Article
ISSN
0002-9483

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✦ Synopsis


Recent reports have suggested that the alpha-1-antitrypsin allele Piz, which in homozygotes results in severe deficiency of this important protease inhibitor, is maintained at a relatively high gene frequency through the mechanism of segregation distortion. We report here on 121 nuclear families selected because only one parent was segregating the Z allele. After correcting for ascertainment, no evidence of preferential transmission was observed in 278 informative offspring.