Aggravation of myasthenia gravis by erythromycin
β Scribed by Dr Eugene F. May; Preston C. Calvert
- Publisher
- John Wiley and Sons
- Year
- 1990
- Tongue
- English
- Weight
- 360 KB
- Volume
- 28
- Category
- Article
- ISSN
- 0364-5134
No coin nor oath required. For personal study only.
π SIMILAR VOLUMES
Amoiridis et al. support this suggestion. Specifically, the thenar-recorded median elbow stimulation (MES)-evoked response has a clear increase in area, but only a minimal concomitant amplitude increase, compared with the median wrist stimulation (MWS)-evoked response. Supramaximal stimulus paramet
Myasthenia gravis (MG) and its animal model experimental autoimmune myasthenia gravis (EAMG) are caused by autoantibodies against nicotinic acetylcholine receptor (AChR) in skeletal muscle. The production of anti-AChR antibodies is mediated by cytokines produced by CD4+ and CD8+ T helper (Th) cells.
## Abstract We report our experience with singleβfiber electromyogrphy (SFβEMG), acetylcholine receptor antibody assays, and Lancaster RedβGreen tests of ocular motility with edrophonium chloride in the diagnosis of myasthenia gravis (MG). Over a period of almost 3 years, 43 patients in whom repeti