Adenosine dialdehyde blocks the disappearance of two nerve growth factor-induced insoluble proteins
β Scribed by Jeb Y. VanDenburg; Philip N. McFadden
- Book ID
- 105390044
- Publisher
- Springer
- Year
- 1995
- Tongue
- English
- Weight
- 953 KB
- Volume
- 14
- Category
- Article
- ISSN
- 1573-4943
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## Abstract Atypical protein kinase Cs zeta and lambda/iota play a functional role in the regulation of NGFβinduced differentiation and survival of pheochromocytoma, PC12 cells [Coleman and Wooten, 1994; Wooten et al., 1999]. Here we demonstrate an NGFβdependent interaction of aPKC with its binding
We have investigated the electrophysiological effects of nerve growth factor (NGF) on single-neuron activity in central nervous system (CNS) grafts of septum, spinal cord, and hippocampus in oculo. NGF was found to have slow-onset, long-lasting excitatory effects on the spontaneous firing of neurons