✦ LIBER ✦

Adenosine A2A-Receptor Antagonism and Pathophysiology of Parkinson’s Disease and Drug-Induced Movement Disorders

✍ Scribed by Kulisevsky, Jaime; Poyurovsky, Michael

Book ID: 118132582
Publisher: S. Karger AG
Year: 2012
Tongue: English
Weight: 503 KB
Volume: 67
Category: Article
ISSN: 0014-3022
DOI: 10.1159/000331768

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

A 2A adenosine receptors and Parkinson's

A 2A adenosine receptors and Parkinson's disease severity

✍ Casetta, I.; Vincenzi, F.; Bencivelli, D.; Corciulo, C.; Gentile, M.; Granieri, 📂 Article 📅 2013 🏛 John Wiley and Sons 🌐 English ⚖ 187 KB

[Handbook of Experimental Pharmacology]

[Handbook of Experimental Pharmacology] Adenosine Receptors in Health and Disease Volume 193 || Adenosine A2A Receptors and Parkinson’s Disease

✍ Wilson, Constance N.; Mustafa, S. Jamal 📂 Article 📅 2009 🏛 Springer Berlin Heidelberg ⚖ 672 KB

Movement Disorders: A Video Atlas Volume

Movement Disorders: A Video Atlas Volume 2 || Parkinson’s Disease: Hoehn and Yahr Scale

✍ Bhidayasiri, Roongroj; Tarsy, Daniel 📂 Article 📅 2012 🏛 Humana Press ⚖ 93 KB

Past, present and future of A2A adenosin

Past, present and future of A2A adenosine receptor antagonists in the therapy of Parkinson's disease

✍ Marie Therese Armentero; Annalisa Pinna; Sergi Ferré; José Luis Lanciego; Christ 📂 Article 📅 2011 🏛 Elsevier Science 🌐 English ⚖ 632 KB

Novel neuroprotection by caffeine and ad

Novel neuroprotection by caffeine and adenosine A2A receptor antagonists in animal models of Parkinson's disease

✍ Anti Kalda; Liqun Yu; Emin Oztas; Jiang-Fan Chen 📂 Article 📅 2006 🏛 Elsevier Science 🌐 English ⚖ 326 KB

Parkinson's disease and LRRK2: Frequency

Parkinson's disease and LRRK2: Frequency of a common mutation in U.S. movement disorder clinics

✍ Denise M. Kay; Cyrus P. Zabetian; Stewart A. Factor; John G. Nutt; Ali Samii; Al 📂 Article 📅 2005 🏛 John Wiley and Sons 🌐 English ⚖ 67 KB 👁 1 views

## Abstract The G2019S mutation in the __LRRK2__ gene is reportedly a common cause of familial Parkinson's disease (PD) and may also have a significant role in nonfamilial PD. The objective of this study was to assess mutation carrier frequency in PD patients from movement disorder clinics in the U