Previous electrophysiological experiments have emphasized the importance of the firing pattern for the functioning of midbrain dopamine (DA) neurons. In this regard, excitatory amino acid receptors appear to constitute an important modulatory control mechanism. In the present study, extracellular re
Activation of nigral dopamine neurons by the selective GABAB-receptor antagonist SCH 50911
β Scribed by S. Erhardt; H. Nissbrandt; G. Engberg
- Publisher
- Springer
- Year
- 1999
- Tongue
- English
- Weight
- 188 KB
- Volume
- 106
- Category
- Article
- ISSN
- 1435-1463
No coin nor oath required. For personal study only.
π SIMILAR VOLUMES
Dopamine D 1 -receptor binding in the basal ganglia is differentially regulated by subtype nonspecific dopamine antagonists such as the antipsychotic, Fluphenazine. The purpose of the present study was to determine the relative contributions of D 1 and D 2 receptor systems in the regulation of basal
The presence of D, dopamine receptors in the parathyroid gland has been proposed based on the demonstration of dopaminergic regulation of adenylate cyclase activity and parathyroid hormone release in dispersed bovine parathyroid cells. Using a radioiodinated D, selective antagonist [12511-SCH 23982,
Extracellular single-unit recording techniques were used to study the effects of the cholecystokinin-A (CCK-A) antagonist, L-364,718, and the CCK-B antagonist, PD 134308, on DA neuronal activity in chloral hydrate anesthetized rats. Neither L-364,718 (0.1-1.6 m g k g i.v.) nor PD 134308 (0.1-6.4 mgk