✦ LIBER ✦

Activated peritoneal macrophages inhibit the proliferation of rat ascites hepatoma AH-130 cells via the production of tumor necrosis factor-αand nitric oxide

✍ Scribed by H. Maekawa; K. Iwabuchi; I. Nagaoka; H. Watanabe; T. Kamano; M. Tsurumaru

Publisher: SP Birkhäuser Verlag Basel
Year: 2000
Tongue: English
Weight: 204 KB
Volume: 49
Category: Article
ISSN: 1420-908X
DOI: 10.1007/s000110050629

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Selenoorganic compound, ebselen, inhibit

Selenoorganic compound, ebselen, inhibits nitric oxide and tumor necrosis factor-α production by the modulation of Jun-N-terminal kinase and the NF-κb signaling pathway in rat Kupffer cells

✍ Naoya Shimohashi; Makoto Nakamuta; Koutaro Uchimura; Rie Sugimoto; Hiroaki Iwamo 📂 Article 📅 2000 🏛 John Wiley and Sons 🌐 English ⚖ 267 KB 👁 1 views

In response to the bacterial endotoxin, LPS, Kupffer cells are induced to express NO and TNF-␣. These compounds are involved in hepatic inflammation/injury, especially that associated with endotoxic shock. In this study, we demonstrate that ebselen (2-phenyl-1,2-benzisoselenazol-3[2H]one), a selenoo

Growth regulation of the AML-193 leukemi

Growth regulation of the AML-193 leukemic cell line: Evidence for autocrine production of granulocyte-macrophage colony-stimulating factor (GM-CSF), and inhibition of GM-CSF-dependent cell proliferation by interleukin-1 (IL-1) and tumor necrosis factor (tnfα)

✍ Vincent Kindler; John Shields; Dominique Ayer; Gonzalo J. Mazzei 📂 Article 📅 1991 🏛 John Wiley and Sons 🌐 French ⚖ 583 KB

The human leukemic cell line AML-193 was tested for its proliferative response to endogenously produced autocrine factors and to a variety of cytokines and colony-stimulating factors. Cells grown in the absence of GM-CSF incorporated tritiated thymidine, and this was partially reversed by adding neu