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Acidosis-induced coronary constriction in the rat heart: Evidence for the activation of L-type calcium channels

✍ Scribed by D. A. Wilson; B. Woodward


Publisher
Springer
Year
1999
Tongue
English
Weight
801 KB
Volume
14
Category
Article
ISSN
0910-8327

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Prion protein fragment 106–126 induces a
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The prion diseases are transmissible neurodegenerative pathologies characterized by the accumulation of altered forms of the prion protein (PrP), termed PrP Sc , in the brain. Previous studies have shown that a synthetic peptide homologous to residues 106-126 of PrP (PrP 106-126) maintains many char