The Dubin-Johnson syndrome is characterized by an patocytes into the bile. 3,4 This defect results in predomiinherited defect in the secretion of amphiphilic anionic nantly conjugated hyperbilirubinemia and a characterconjugates from hepatocytes into the bile. We have reistic secondary rise of intra
โฆ LIBER โฆ
Absence of the canalicular isoform of the MRP gene-encoded conjugate export pump from the hepatocytes in Dubin-Johnson syndrome
โ Scribed by J Kartenbeck; U Leuschner; R Mayer; D Keppler
- Book ID
- 118686160
- Publisher
- John Wiley and Sons
- Year
- 1996
- Tongue
- English
- Weight
- 603 KB
- Volume
- 23
- Category
- Article
- ISSN
- 0270-9139
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cMRP IN HUMAN LIVER AND THE ABSENCE OF cMrp The human Dubin-Johnson syndrome and its animal OR cMoat IN CANALICULAR MEMBRANES OF TR 0 TRmodel, the TR 0 rat, are characterized by a chronic conju-RAT HEPATOCYTES gated hyperbilirubinemia. TR 0 rats are defective in the Mayer R, Kartenbeck J, Bu ยจchler
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