The extracerebral vasculature receives a postnatal innervation of noradrenergic sympathetic axons and nociceptive sensory axons. These axons are responsive to the neurotrophin nerve growth factor (NGF), in that they possess the transmembrane receptors p140 proto-trkA and p75 neurotrophin receptor (N
Absence of p75NTR expression reduces nerve growth factor immunolocalization in cholinergic septal neurons
β Scribed by Karmen M. Krol; Keith A. Crutcher; Bettina E. Kalisch; R. Jane Rylett; Michael D. Kawaja
- Publisher
- John Wiley and Sons
- Year
- 2000
- Tongue
- English
- Weight
- 449 KB
- Volume
- 427
- Category
- Article
- ISSN
- 0021-9967
No coin nor oath required. For personal study only.
π SIMILAR VOLUMES
Epithelial expression of the 75-kDa low-affinity neurotrophin receptor (p75 NTR ) is inversely associated with the malignant progression of the human prostate. To elucidate the function of p75 NTR in the prostate, the human prostate epithelial tumor cell line TSU-pr1, which does not express p75 NTR
Nerve growth factor (NGF) is a potential treatment for cholinergic dysfunction associated with Alzheimer's disease (AD). In rats, NGF activates gene expression of the acetylcholine synthetic enzyme choline acetyltransferase (ChAT) and prevents age-and lesion-induced degeneration of basal forebrain (
Acetylcholine (ACh) is synthesized by choline acetyltransferase (ChAT) in the cytoplasm of cholinergic nerve terminals and transported into synaptic vesicles by vesicular ACh transporter (VAChT). The genes encoding ChAT and VAChT are colocalized within the genome, and their products are known to be