𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Abnormal osteoclast morphology and bone remodeling in a murine model of a lysosomal storage disease

✍ Scribed by M.A Monroy; F.P Ross; S.L Teitelbaum; M.S Sands

Book ID
117807557
Publisher
Elsevier Science
Year
2002
Tongue
English
Weight
898 KB
Volume
30
Category
Article
ISSN
8756-3282

No coin nor oath required. For personal study only.

πŸ“œ SIMILAR VOLUMES

Crosstalk of osteoblast and osteoclast p
Crosstalk of osteoblast and osteoclast precursors on mineralized collagenβ€”towards an in vitro model for bone remodeling
✍ A. Bernhardt; S. Thieme; H. Domaschke; A. Springer; A. RΓΆsen-Wolff; M. Gelinsky πŸ“‚ Article πŸ“… 2010 πŸ› John Wiley and Sons 🌐 English βš– 617 KB

## Abstract Bone remodeling and, therefore, integration of implant materials require the coordinated regulation of osteoblast and osteoclast activity. This is why the __in vitro__ evaluation of biomaterials for bone regeneration should involve not only the analysis of osteoblast differentiation but

Minimum requirement of donor cells to re
Minimum requirement of donor cells to reduce the glycolipid storage following bone marrow transplantation in a murine model of Fabry disease
✍ Takayuki Yokoi; Hiroshi Kobayashi; Yohta Shimada; Yoshikatsu Eto; Nobuyuki Ishig πŸ“‚ Article πŸ“… 2011 πŸ› John Wiley and Sons 🌐 English βš– 229 KB

## Abstract ## Background Fabry disease (FD) is a lysosomal storage disorders characterized by a deficiency of the lysosomal enzyme, α‐galactosidase A. This results in the accumulation of glycolipids, mainly globotriaosylceramide (GL‐3), in the lysosomes of various organs. Although bone marrow tra