𝔖 Bobbio Scriptorium
✦   LIBER   ✦

A pharmacological study of changes in central nervous system receptor responsiveness after long-term dexamphetamine and apomorphine administration

✍ Scribed by Ruth C. Bailey; D. M. Jackson

Publisher
Springer
Year
1978
Tongue
English
Weight
950 KB
Volume
56
Category
Article
ISSN
0033-3158

No coin nor oath required. For personal study only.

πŸ“œ SIMILAR VOLUMES

The demonstration of a change in adrener
The demonstration of a change in adrenergic receptor sensitivity in the central nervous system of mice after withdrawal from long-term treatment with haloperidol
✍ Robin Dunstan; David M. Jackson πŸ“‚ Article πŸ“… 1976 πŸ› Springer 🌐 English βš– 935 KB

Mice, administered haloperidol (3 mg/kg/d) in their drinking water for 21 days, displayed, 4 days after cessation of the haloperidol-treatment, marked locomotor stimulation to clonidine (100 or 500 mug/kg) which lasted for about 6 h. 25 mug clonidine/kg was inactive. Premedication with FLA-63 (25 mg

Effect of long-term vigabatrin therapy o
Effect of long-term vigabatrin therapy on GABA and other amino acid concentrations in the central nervous system β€” a case study
✍ Elinor Ben-Menachem; Anders Hamberger; John Mumford πŸ“‚ Article πŸ“… 1993 πŸ› Elsevier Science 🌐 English βš– 296 KB

A 32 year old patient with refractory complex partial seizures was treated with vigabatrin for 3.5 years. Before starting treatment and at 42 months. lumbar punctures were done and the CSF analyzed for amino acids including GABA. Although the patient experienced a 50% seizure reduction, he underwent

Early postnatal chronic inflammation pro
Early postnatal chronic inflammation produces long-term changes in pain behavior and N-methyl-D-aspartate receptor subtype gene expression in the central nervous system of adult mice
✍ Joan M.C. Blom; C. Benatti; S. Alboni; G. Capone; C. Ferraguti; N. Brunello; F. πŸ“‚ Article πŸ“… 2006 πŸ› John Wiley and Sons 🌐 English βš– 442 KB

## Abstract The objective of this study was to test whether postnatal chronic inflammation resulted in altered reactivity to pain later in life when reexposed to the same inflammatory agent and whether this alteration correlated with brain‐region‐specific patterns of N‐methyl‐D‐aspartate (NMDA) rec