Fibril formation is the hallmark of pathogenesis in Alzheimer's disease and other amyloid disorders caused by conformational alterations leading to the aggregation of soluble monomers. AΞ²40 selfβassociates to form amyloid fibrils. Its central sevenβresidue segment KLVFFAE (AΞ²16β22), which is thought
β¦ LIBER β¦
A Novel Technique for the Effective Production of Short Peptide Analogs from Concatameric Short Peptide Multimers
β Scribed by Sang Jun Lee; Jong Hee Lee; Hyun Joo Jin; Jeong Ho Lee; Ho Young Ryu; Yoon Kim; In Soo Kong; Kyu Won Kim
- Book ID
- 106262829
- Publisher
- Korean Society for Molecular and Cellular Biology
- Year
- 2000
- Tongue
- English
- Weight
- 99 KB
- Volume
- 10
- Category
- Article
- ISSN
- 1016-8478
No coin nor oath required. For personal study only.
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