Background: IBD5, IL23R, and ATG16L1 genetic variations are established Crohn's disease (CD) risks alleles. We evaluated these in a population-based case-control study within a cohort to determine their penetrance, population attributable risk, independence, and relationship to other established CD
A Nonsynonymous SNP in ATG16L1 Predisposes to Ileal Crohn’s Disease and Is Independent of CARD15 and IBD5
✍ Scribed by Natalie J. Prescott; Sheila A. Fisher; Andre Franke; Jochen Hampe; Clive M. Onnie; Dianne Soars; Richard Bagnall; Muddassar M. Mirza; Jeremy Sanderson; Alastair Forbes; John C. Mansfield; Cathryn M. Lewis; Stefan Schreiber; Christopher G. Mathew
- Book ID
- 119759757
- Publisher
- Elsevier Science
- Year
- 2007
- Tongue
- English
- Weight
- 246 KB
- Volume
- 132
- Category
- Article
- ISSN
- 0016-5085
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