3Impact of clinical and nutritional parameters on the outcome of familial amyloid polyneuropathy after liver transplantation
β Scribed by BITTENCOURT, P; COUTO, C; LALLEE, M; FARIAS, A; MASSAROLLO, P; RAIA, S; MIES, S
- Book ID
- 122520933
- Publisher
- John Wiley and Sons
- Year
- 2000
- Tongue
- English
- Weight
- 146 KB
- Volume
- 6
- Category
- Article
- ISSN
- 1527-6465
No coin nor oath required. For personal study only.
π SIMILAR VOLUMES
To elucidate whether the amount of tissue-deposited amyloid in familial amyloid polyneuropathy (FAP) patients decreases or increases over the long-term course after liver transplantation (LT), we examined changes in histopathological and biochemical characteristics of abdominal fat amyloid in the tr
Familial amyloid polyneuropathy (FAP) is a form of hereditary generalized amyloidosis. Liver tissue explanted from FAP patients has normal structure and function, except for the production of amyloidogenic variant transthyretin (TTR), and domino liver transplantation (DLT) using grafts from FAP pati
Familial amyloidotic polyneuropathy (FAP) patients present adrenergic cardiac input blockade secondary to amyloid deposits and sympathetic neuropathy. Consequently, their capacity to compensate for hemodynamic changes is limited. To avoid hemodynamic disturbances in sequential liver transplants, a s